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Extrinsic activation involved the dysregulation of transporter proteins, including transferrin (Tfrn1) and cystine-glutamate antiporter (Xc-) on the plasma membrane, and mitoferrin (Mfrn) on the IMM (152)

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These rapid changes in LPL activity are determined by post-translational mechanisms that involve interactions with proteins of the angiopoietin-like protein family as well as with apolipoproteins in the circulating chylomicrons and VLDL