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Moreover, Genistein, by reduction of pro-inflammatory mediators (IL-6, tumor necrosis factor- (TNF-) and C-reactive protein (CRP)) in serum and intestine in alloxan-induced diabetic rats, reduces the damage of L-cells and leads to an increase in the expression and secretion of GLP-1[191]

Journal of Metabolic Medicine, 12 (6), 512523

M., Singh, S., Segal, J

Compared with disulfide-based systems, which are widely used due to their simplicity and predictable cleavage by intracellular GSH, diselenide linkages offer greater redox sensitivity and can respond to both GSH and ROS

X-linked or partial defects can be unmasked by stress (e.g., sepsis, critical illness, steroids, fasting, surgery, pregnancy)