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What emerges is a coherent biological framework: estrogen maintains the mitochondrial infrastructure and synaptic stability that neurons depend on for sustained function, while GLP-1 receptor activation reduces the inflammatory burden that progressively degrades that infrastructure

The LactMed database states that tirzepatide use is not a reason to discontinue breastfeeding

These findings identify ALC as a metabolic regulator that restores social isolation-induced myelin deficits by coordinating myelin protein synthesis, mitochondrial function, and myelin lipid remodeling