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Finally, data from mouse studies show that IgG accumulates in white adipose tissue in an FcRn-dependent manner, and that knocking down the receptor by an antisense oligonucleotide targeting the encoding FCGRT gene or deleting expression of FcRn from myeloid cellseffectively turning macrophages into degradative sinks for IgG [381]surprisingly improves metabolic function in mice with diet-induced obesity [382]

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Baseline assessment should include fasting IGF-1, growth hormone, cortisol, prolactin, and complete metabolic panel

J Clin Microbiol 47(4):959 Hatakeyama M (2011) Anthropological and clinical implications for the structural diversity of the Helicobacter pylori CagA oncoprotein

In the muscle atrophy mouse model, Ex-4 also increased muscle mass and muscle fiber size and improved muscle function