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These transposable elements include transposons and retrotransposons, such as endogenous retroviruses, which can essentially cut and paste into any portion of the human genome
The hypothesis that deficient serotonergic neurotransmission contributes to the pathogenesis of MDD originated from clinical observations that amine-depleting drugs induced depressive states, whereas monoamine oxidase inhibitors, which prevent the breakdown of serotonin (5-HT), alleviated depressive symptoms [4, 5]
these vials are NOT for injection.
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