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This is mainly due to the fact that research has traditionally focused on well-known stress molecules like SA or JA, or simply measured the activity of selected redox enzymes
The most harmful scenario is having gene variants that reduce glutathione levels and high toxin levels that deplete glutathione
Elevation of the ratio of Cu/Znsuperoxide dismutase to glutathione peroxidase activity induces features of cellular senescence and this effect is mediated by hydrogen peroxide
Redox compartmentalization in eukaryotic cells
Not only that, the maximum safe discharge rate is a moving target that will fluctuate depending on several factors including pack temperature, age and internal resistance (more on this in a bit)
