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By activating two distinct but complementary pathways, the combination triggers a much more significant and controlled GH release in lab models than either peptide could ever manage on its own
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Targeting mitochondrial dynamics and metabolic flux regulation offers a novel strategy to disrupt cancer cell metabolic plasticity and enhance therapeutic efficacy [46]
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Membrane damage during ferroptosis is caused by oxidation of phospholipids catalyzed by the oxidoreductases POR and CYB5R1
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