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The liver uses glutathione for detoxification, but excessive consumption can overwhelm this process leading to depletion and increased oxidative stress

N-Acetyl-Cysteine (NAC)is a source of the conditionally essential amino acid L-cysteine and a precursor to the tripeptide glutathione

These modifications were designed to reduce binding affinity for IGF binding proteins (IGFBPs), which normally sequester circulating IGF-1 and limit its bioavailability

University pharmacists dispensed either active supplements or placebo at the baseline visit, and subjects were provided detailed instructions for supplement usage

Additionally, these metabolic interventions can potentially reverse the immunosuppressive phenotype of the TME, enhancing the efficacy of ICIs [77, 78]