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Clinically, TIND can manifest with prominent lower extremity weakness while characteristically preserving ankle reflexes, and it has also been reported to produce diabetic lumbosacral radiculoplexus neuropathy characterized by painful asymmetric weakness, findings that parallel the presentation observed in this patient [7,8,9]

593 Reactive astrocytes play predominantly protective role, and inhibition of reactivity exacerbates pathology in AD animal models

SCFAs stimulate the L-cells and release GLP-1[8]

Resistance is promoted by a range of factors that are either present in the tumour before therapy (described as intrinsic resistance) or selected for during therapeutic exposure (termed acquired resistance)

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