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UPR activation induced by enhanced TCA flux could enhance the functionality of the organelle in anticipation of the increase in protein synthesis including synthesis of ER client proteinsthat follows in the post-absorptive state

Although the mechanism(s) underlying NASH progression remain to be fully elucidated, accumulating evidence suggests that both oxidative stress and the pro-inflammatory cytokine tumor necrosis factor- (TNF) are critical contributors in this process 5

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