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As GLP-1R agonists mediate their appetite-reducing effects within the central nervous system, the trafficking phenotype of exendin-phe1 could result in reduced brain penetration, mitigating against presumed advantageous effects on neuronal GLP-1R desensitization
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In over 500 preclinical studies, it has demonstrated regenerative effects on tendons, ligaments, muscles, the gastrointestinal tract, and the peripheral nervous system
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