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These findings suggest that neither direct release of GLP-1 in the OB from hindbrain PPG neuron projections, nor volume transmission via release of GLP-1 into the ventricles are likely sources of GLP-1 to activate OB GLP-1R
however, there was no effect on plasma insulin
And some of it is valid
Researchers are driven by a deep curiosity to answer fundamental questions: Can we mitigate the effects of cellular senescence
[3] Nikolaeva et al
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