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Sources conflict on the exact relationship: some describe fosgonimeton as a phosphate prodrug of Dihexa, while Athira has described a structurally distinct molecule so it is most accurate to call it the same program, structurally distinct, with conflicting public descriptions

This further worsens glycaemic control in these patients, and in combination with the reduced insulinotropic potency renders GIP an undesirable therapeutic target for T2D treatment